There had been no need for ocular corticosteroids for 3 months. Three months after her initial visit, the patient reported that her blurry vision had resolved and her fatigue had improved. In between the visits, the patient had her eyes examined by her ophthalmologist and was told that her eye pressure had normalized. At this point, we also added reduced glutathione (100 mg BID) to her regimen, to support the small blood vessels in the eyes. She was instructed to continue on the regimen as previously described.
#Kaleidoscope vision treatment skin
Her skin lesions on the feet and scalp were thinner, smaller, less erythematous, and not itchy. She was tolerating the thyroid medication well. She also mentioned that a feeling of eye fullness of several months’ duration was now improving as well. Four-Week Follow-upįour weeks later, the patient reported better energy, less blurriness, and less pain in her eyes. She also was prescribed calcipotriene 0.005% ointment, to apply BID to the affected areas on her skin. She was started on dessicated thyroid (30 mg QAM), vitamin D3 (10 000 IU/d), and iron glycinate (34 mg QD). The patient was diagnosed with microcytic anemia, hypothyroidism, and severe vitamin D3 deficiency. Lab test results were received in 5 days, at which point the patient returned for a follow-up visit. The patient was advised to start on bilberry extract, 160 mg BID.
#Kaleidoscope vision treatment free
I ordered the following laboratory tests : CBC, TSH, free T4 and free T3 levels, vitamin D (25-hydroxyvitamin D), and a renal panel. Her optic discs were clear, but small fundal blood vessel crowding was observed. Her ophthalmologic exam revealed injected sclerae and normal and symmetric pupillary reaction to light. Both the foot lesions and the scalp lesions had a clinical appearance of plaque psoriasis. The lesions were scaling, cracking, thick, and erythematous. These were previously diagnosed as athlete’s foot. She had painful red lesions on the bottoms of her feet, bilaterally. Physical exam revealed the following: BP, 92/56 mm Hg temperature, 96.7° F, cervical lymphadenopathy non-tender thyromegaly mild inspiratory wheezes on lung auscultation systolic heart murmur and tender splenomegaly on abdominal examination. We reviewed prior imaging tests, including head CT scan and MRI of the brain, neither of which showed brain lesions. Due to her vision decline, she had taken a medical leave of absence from her work.
At the time of consultation, the patient could barely function and was unable to perform her daily activities. Her vision returned but had since become blurry everything was described as being “behind a veil.”įatigue had been gradually developing, and she had a long history of anemia. She was diagnosed with ocular inflammation and treated with ocular corticosteroids. Several weeks prior to her visit, she started experiencing sudden temporary loss of vision in both eyes. She had not had glaucoma, but her family history (mother, maternal grandmother) was positive for acute narrow angle glaucoma. She was under the care of an ophthalmologist for monitoring of increased intraocular pressure. The photophobia had been present for several years, and she was used to wearing sunglasses, even indoors and during winter months. Case StudyĪ 39-year-old female presented for a naturopathic consultation with chief complaints of blurry vision, fatigue, and photophobia. The following case study describes a correlation between sudden temporary loss of vision, blurry vision, chronic photophobia, and severe vitamin D deficiency. Vitamin deficiencies have long been implicated in gradual decline of vision however, an association between sudden vision loss and nutritional deficiencies has been less well researched. In most cases of sudden vision loss, head/eye trauma, or an autoimmune neurologic condition (such as multiple sclerosis) is the underlying cause. Sudden visual disturbances and partial loss of vision are usually quite disabling symptoms that make an affected individual seek prompt medical care.
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